Advances in Mercury Toxicology by Tsuguyoshi Suzuki, Nobumasa Imura, Thomas W. Clarkson

By Tsuguyoshi Suzuki, Nobumasa Imura, Thomas W. Clarkson

This e-book is predicated on a world assembly equipped by way of the collage of Tokyo and the collage of Rochester, and is released as one belonging to the sequence of Rochester overseas meetings in Environmental Toxicity. The assembly on "Advances in Mercury Toxicology" used to be held on the college of Tokyo on August 1 to three, 1990. The invited papers are released during this ebook besides an "Overview" bankruptcy that used to be written through the editors at a gathering held on the collage of Rochester on August 1 to two, 1991. the aim of the assembly used to be to gather prime scientists to debate their most up-to-date findings at the toxicology of mercury. The time was once opportune. enormous growth has been made at the environmental destiny and toxicology of mercury. contemporary findings have given new perception into the worldwide version for mercury. delivery within the surroundings extends nice distances leading to toxins of lakes and rivers a long way far-off from the resource of mercury free up. the method of methylation ends up in accumulation of methylmercury in fish and therefore within the human vitamin. New facts shows that acid rain and the impoundment of water for hydroelectric reasons impacts the methylation and bioaccumulation approaches leading to better degrees of methylmercury in fish.

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Congenital Minamata Disease due to prenatal exposure is characterized by diffuse damage to the central nervous system. A history of the mother's exposure during pregnancy is useful in diagnosis. The umbilical cord is frequently preserved in Japanese families and this can provide evidence of prenatal exposure. Studies in other populations exposed to methylmercury were briefly discussed by Skerfving. Infants of mothers with exposures considerably lower than those seen in the severe cases in Minamata exhibited possible evidence of slight retardation of central nervous function.

Most of this retained dose passes immediately into the bloodstream, the remainder being deposited in lung tissue. Mercury vapor can also cross the skin. Quantitatively, transport across the skin is much less important than pulmonary uptake in people exposed to mercury vapor. On entering the blood compartment, it dissolves in plasma as the monatomic gas. It is lipid soluble and highly diffusible. It rapidly leaves the plasma to enter the red cells. It does, however, persist in plasma for a sufficient period to circulate to all parts of the body.

Another type of renal injury, glomerular injury, was least understood until the effect of mercury on the immune system had been intensively studied with several experimental models which allowed an elucidation of the mechanism (see chapter by Druet). Various mercurials may induce autoimmune glomerular lesions, and other pathological conditions such as pink disease and possibly Kawasaki disease, an acute febrile mucocutaneous syndrome. The two diseases may involve disturbances in the immune system by mercury.

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